From the symptoms you briefly described, I recommend that you consider acne. Here are some relevant information, I hope it will be helpful to you. The type of acne lesion, minimal acne, is considered to be a precursor of acne vulgaris. The clinical lesions of acne vulgaris include closed acne, open acne and inflammatory papules, pustules and nodules. The process by which micro-acne develops into other acne lesions has not been clarified, but may involve the following aspects: the accumulation of sebum and keratinizing substances transforms micro-acne into closed acne (white-headed acne). The follicular orifice is open due to continuous expansion, forming open acne (blackhead acne). Closely packed keratinocytes, oxidized lipids and melanin all make open acne dark. The destruction of hair follicles promotes the development of inflammatory lesions. After the destruction of hair follicles, pro-inflammatory lipids and keratins are squeezed into the surrounding dermis, leading to the formation of inflammatory papules and nodules.
Androgenesis - For patients with acne and other signs of androgenesis, laboratory tests for androgenesis are required. PCOS is the most common cause of androgenism in women. The main features of the disease are menstrual disorders, hairy, acne, ovarian cysts, insulin resistance to varying degrees, and acanthosis nigra. The rapid onset of acne combined with feminization suggests potential adrenal or ovarian tumors. Acne vulgaris may also occur in patients with Cushing's disease or Cushing's syndrome and delayed congenital adrenocortical hyperplasia. Evidence of feminization includes low voice, reduced breast size, hypertrophy of clitoris, hair loss, hypomenorrhea and hirsutism. These patients may need imaging examinations of the adrenal glands and ovaries as well as hormone assessment. In these cases, referral to the endocrinology department should be considered. Preliminary screening for androgenism is recommended. The levels of DHEA-S, total testosterone and free testosterone were all rea
